Ph.D. position in molecular immunology
We are looking to recruit a Ph.D. student in molecular immunology to study innate immune pathways leading to autoinflammation. The selected candidate will be enrolled in the international Ph.D. program in cancer and immunology @UNIL.
The position is funded and available as soon as possible; however, the starting date can be arranged.
The ideal candidate must be enthusiastic, passionate, and deeply interested in deciphering the molecular mechanisms of signaling pathways such as the inflammasome and their contribution to human diseases. In addition, the candidate must have an inquiring mind, willing to ask questions and challenge the findings.
The candidate should have a Master's degree in biology or a related field.
The applicant should have good communication skills in English and be able to express and organize ideas in a meaningful way.
Please, send an email with a CV and motivation letter to Fabio.Martinon@unil.ch
Fabio Martinon received his PhD in 2003 from the University of Lausanne for his work on the characterization of the Inflammasome in the laboratory of Jürg Tschopp. After a short post-doctoral fellowship in Jürg Tschopp’s laboratory, he moved in 2006 to the laboratory of Laurie Glimcher at the Harvard School of Public Health, where he investigated the link between inflammatory programs and the endoplasmic reticulum stress response. In August 2010 he joined the Department of Biochemistry as Assistant Professor and Associate Professor in 2017. His current research focuses on the characterization signaling pathways leading to inflammation.
Cellular stress is a hallmark of severe pathogenic insults that leads to the activation of adaptation programs aimed at restoring tissue homeostasis. This state of reversible malfunction is defined by the notion of stress. Tissue stress and malfunction promotes low-grade inflammation that helps the tissue to cope with damage and restore tissue function. This low-grade inflammation also termed para-inflammation illustrates how stress and possibly stress response pathways can regulate immunity. A variety of diseases are characterized by inflammation and cellular homeostasis deregulation including viral infections, cancer and autoinflammatory syndromes. However, the mechanisms and the role of these stress responses and their impact on the inflammatory pathways are poorly understood. We developed a research program aimed at identifying stress-signaling pathways that regulate inflammation. Our research is aimed at characterizing the stress-inflammation connection in autoinflammatory diseases and cancer.
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